| Autism
Children
who are autistic
have a lifelong developmental disability, but the degree of symptoms
and
problems that they have can vary from mild to severe. The symptoms
begin prior
to the third birthday, and are most commonly noticed around the 30th
month. Among
the problems that autistic children may have include
difficulty
interacting socially with other and developing normal social skills.
This can
cause difficulty developing age appropriate social relationships with
family
and peers, an inability to use nonverbal social interactions, such as
returning
a smile or making eye contact, they may become attached to unusual
objects
(such as a rubber band or a stick) and they may have no interest in
sharing
their interests, achievements or things they enjoy with others. Other
early
symptoms can include not developing a social smile, being overly
irritable and
easily upset, not cuddling, being indifferent to parents, not playing
social
games (such as peek-a-boo), and using people as tools.
Autism
is a severe mental disorder
beginning in infancy characterized by impairment of mutual social
interaction
(neglecting the existence of feelings in others), of verbal and
nonverbal
communication, and restricted repertoire of activities and interest.
These
children are often self absorbed, unreachable and unable to relate to
others.
Temper tantrums occur if the child is interrupted. Childhood autism is
a
behavioural syndrome with several different aetiologies. Its prevalence
is
estimated at about one in every 1000 children born. The male: female
ratio is
considerably higher than in the general population, but possibly not as
high as
3-4:1. Genetic factors contribute to the development of autism in some
cases.
Defects
in respiratory chain
enzymes, Pyrurate Dehydrogenase or mitochondrial respiratory chain
defects,
that is, NAD, NADH, CoEnzyme Q10 and cytochrome oxidase deficiency. Although
we
find a variety of autistic phenotypes to have associated mitochondrial
abnormalities, the most common nonspecific PDD, typically of a form
that
manifests language and cognitive regression or stagnation during the
second
year. Most surprising among multiplex families is that the biochemical
and
clinical marker of mitochondrial disease often segregate in an
autosomal
dominant manner (that is, genetically induced). Although no molecular
lesion
has yet been found in the NAD/NADH activity, the biochemical findings
are most
consistent with abnormal mitochondrial complex 1 activity (that is
NAD/NADH
activity). Early and careful evaluation of autistic children for these
more
subtle mitochondrial disturbances may rescue them from more severe
brain
injury.
Relevancy
for NAD Energy supplementation in autism:
The
average NAD Energy Block value found in patients suffering from
alcoholism is 70, where 100 is the borderline value.
Steps to
treatment of NAD Energy Deficiency
NAD
Energy supplements are preferred to be used in co-operation with your
health care professional, although you may choose to follow a self help
approach in treating NAD Energy Deficiency.
Click
here to view the medically pre formulated NAD Energy Supplementation
Protocols for autism
Medically
pre formulated treatment protocols for female alcoholics based upon the
experience of NutriNAD prescribing practitioners and feedback from
regular NutriNAD users.
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